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Showing results 1 to 8 of 8
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Publication Date2020-03
A DLG2 deficiency in mice leads to reduced sociability and increased repetitive behavior accompanied by aberrant synaptic transmission in the dorsal striatum
MOLECULAR AUTISM, v.11, no.1, pp.19 - 19
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Publication Date2019-04
Early Correction of N-Methyl-D-Aspartate Receptor Function Improves Autistic-like Social Behaviors in Adult Shank2−/− Mice
BIOLOGICAL PSYCHIATRY, v.85, no.7, pp.534 - 543
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Publication Date2018-10
GABA neuronal deletion of Shank3 exons 14-16 in mice suppresses striatal excitatory synaptic input and induces social and locomotor abnormalities
FRONTIERS IN CELLULAR NEUROSCIENCE, v.12, pp.341
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Publication Date2016-05
IQ Motif and SEC7 Domain-containing Protein 3 (IQSEC3) Interacts with Gephyrin to Promote Inhibitory Synapse Formation
JOURNAL OF BIOLOGICAL CHEMISTRY, v.291, no.19, pp.10119 - 10130
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Publication Date2018-06
NGL-2 Deletion Leads to Autistic-like Behaviors Responsive to NMDAR Modulation
CELL REPORTS, v.23, no.13, pp.3839 - 3851
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Publication Date2018-09 Sexually dimorphic behavior, neuronal activity, and gene expression in Chd8-mutant mice
NATURE NEUROSCIENCE, v.21, no.9, pp.1218 - 1228
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Publication Date2019-10
Shank3 Exons 14–16 Deletion in Glutamatergic Neurons Leads to Social and Repetitive Behavioral Deficits Associated With Increased Cortical Layer 2/3 Neuronal Excitability
FRONTIERS IN CELLULAR NEUROSCIENCE, v.13, pp.458
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Publication Date2019-06
Shank3 Mice Carrying the Human Q321R Mutation Display Enhanced Self-Grooming, Abnormal Electroencephalogram Patterns, and Suppressed Neuronal Excitability and Seizure Susceptibility
FRONTIERS IN MOLECULAR NEUROSCIENCE, v.12, pp.155
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